Dyspareunia in Women
Dyspareunia is persistent or recurrent genital pain before, during or after sexual intercourse. It is common but frequently underreported, as many women feel substantial embarrassment and reluctance in raising it.
It is classified by anatomical location. Superficial (entry) dyspareunia reflects pathology at or near the vaginal introitus. Deep dyspareunia reflects pathology deeper within the pelvis.
This distinction directs the likely causes and the diagnostic and management approach. Assessment must be sensitive and thorough, given the frequently significant impact on quality of life, sexual function and intimate relationships.
Classification and pathophysiology
- Superficial dyspareunia most commonly reflects genitourinary syndrome of menopause (vulvovaginal atrophy), a particularly common cause in postmenopausal women.
- Other causes of superficial dyspareunia are vulvodynia, vulvovaginal infection (including candidiasis and other infective causes) and, less commonly, vulvar dermatological conditions (including lichen sclerosis).
- Vaginismus is involuntary spasm of the pelvic floor and vaginal musculature. It is frequently associated with psychological or relational factors, including previous painful intercourse establishing a self-perpetuating pain-anxiety-spasm cycle or, in some cases, previous sexual trauma.
- Deep dyspareunia most commonly reflects endometriosis, a particularly important and common cause. Pain is often related to the position of penetration, given involvement of the posterior compartment or uterosacral ligament.
- Other causes of deep dyspareunia are pelvic inflammatory disease and its sequelae (including chronic pelvic adhesions), uterine fibroids, ovarian pathology and pelvic floor dysfunction. These causes frequently coexist and contribute in combination.
- Psychological and relational factors contribute across both superficial and deep dyspareunia. They include anxiety, a history of sexual trauma and relationship difficulty.
- In a self-perpetuating cycle, anticipated pain produces protective pelvic floor muscle tension, which exacerbates the pain. Assess these factors sensitively alongside evaluation for any organic cause.













