Posterolateral Corner Injury
Also known as: PLC Injury, Posterolateral Knee Injury
Posterolateral corner (PLC) injury is a disruption of the structures on the posterolateral side of the knee that resist varus force, external rotation and posterior translation. Its main structures are the lateral collateral ligament (LCL), the popliteus tendon and the popliteofibular ligament, with secondary contributions from the biceps femoris, the lateral capsule, the fabellofibular ligament and the iliotibial band.
It follows a varus force on the knee, a hyperextension injury, a posterolateral blow to the flexed tibia, or a knee dislocation, in road traffic collisions, sports (football, rugby, skiing) and falls. Isolated PLC injury is uncommon (under 30%); most are combined with posterior cruciate ligament (PCL) or anterior cruciate ligament (ACL) tears.
The Fanelli classification describes the instability: type A is an isolated rotational (posterolateral) instability, type B is combined rotational and varus instability, and type C is complete instability with varus and rotational laxity. Grading is by the lateral joint line gap on varus stress: grade I under 5 mm, grade II is 5 to 10 mm, and grade III is over 10 mm.
Complications are chronic posterolateral instability, failed cruciate reconstruction when the PLC is missed, common peroneal nerve palsy (15 to 29%), popliteal artery injury (with knee dislocation), arthritis, and varus thrust gait.
Pathophysiology.
The LCL resists varus stress at 30 degrees of flexion. The popliteus tendon and popliteofibular ligament resist external rotation of the tibia, particularly at 30 to 90 degrees of flexion. The PLC also acts with the PCL to resist posterior translation, so combined injuries cause greater instability and place heavy loads on any cruciate graft.
The common peroneal nerve runs behind the biceps femoris tendon and around the fibular neck, where it is stretched or torn by varus force.













